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Can-CCataractDogs
Treatment with 1% N-acetylcarnosine (NAC) protects telomeres to prevent and dissolve mature cataracts in dogs.

Cataracts in small animals are caused by oxidative stress on lens epithelial cells. Treatment with 1% N-acetylcarnosine (NAC) releases L-carnosine ...

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Can-CDry EyeNac-C Plus
Study finds that Dry Eye Disease (DED) progression may be delayed by breaking the chronic cycle of ocular surface inflammation

The purpose of this article is to review the evidence for the hypothesis that the core mechanism of Dry Eye ...

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CataractDry EyeN-acetylcarnosine
N-alpha-Acetylcarnosine is proposed for treatment of ocular disorders that have a component of oxidative stress in their genesis

Nalpha-acetylcarnosine is proposed as a prodrug of L-carnosine that is resistant to enzymatic hydrolysis by carnosinase with ability to give ...

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Can-CGlaucomaNac-C Plus
Oxidative stress in the human trabecular meshwork clinically correlated with primary open-angle glaucoma

Study evaluated intensity of oxidative molecular damage and its clinical correlation to intraocular pressure. It found that oxidative stress could ...

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Can-CCataractDogs
The role of free radical-induced lipid oxidation in the development of cataracts in human and canine eyes

Free radical-induced lipid oxidation causes accumulation of lenticular lipid peroxidation (LPO) products. Lens opacity correlates with the level of the ...

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Can-CGlaucomaNac-C Plus
Mitochondrial complex I defect induces ROS release and degeneration in trabecular meshwork cells of POAG patients: protection by antioxidants

The authors present strong evidence that mitochondrial complex I defects are the major source of oxidative stress in TM cells ...

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